This day, August 8th, 1794, I have seen a negro, who was born (as he reports) of black parents, both father and mother, at Kingston in Jamaica, who has many large white blotches on the skin of his limbs and body; which I thought felt not so soft to the finger, as the black parts. He has a white divergent blaze from the summit of his nose to the vertex of his head; the upper part of which, where it extends on the hairy scalp, has thick curled hair, like the other part of his head, but quite white. By these marks I supposed him to be the same black, who is described, when only two years old, in the Transactions of the American Philosophical Society, Vol. II. page 292, where a female one is likewise described with nearly similar marks.

The joining of the frontal bones, and the bregma, having been later than that of the other sutures of the cranium, probably gave cause to the whiteness of the hair on these parts by delaying or impeding its growth.

ADDITION [II].

The following extract from a letter of Dr. Beddoes on hydrocephalus internus, I esteem a valuable addition to the article on that subject at Class [I. 2. 3. 12].

"Master L——, aged 9 years, became suddenly ill in the night about a week before I saw him. On the day before the attack, he had taken opening medicines, and had bathed afterwards. He had complained of violently acute pain in his head, shrieked frequently, ground his teeth hard, could not bear to have his head raised from the pillow, and was torpid or deaf. His tongue was white, pulse 110 in the evening and full. As yet the pupil of the eye was irritable, and he had no strabismus. He had been bled with leeches about the head, and blistered. I directed mercurial inunction, and calomel from 3 to 6 grains to be taken at first every six, and afterwards every three hours. This plan produced no sensible effect, and the patient died on the 18th day after the seizure. He had convulsion fits two days preceding his death, and the well-known symptoms of hydrocephalus internus all made their appearance. From what I had seen and read of this disease, I believed it to belong to inflammations, and at an earlier period I should be tempted to bleed as largely as for pneumonia. The fluid found after death in the ventricules of the brain I impute to debility of the absorbents induced by inflammation. My reasons are briefly these; 1. The acuteness of the pain. 2. The state of the pulse. In the above case for the first 9 or 10 days it did not exceed 110, and was full and strong.

3. To find out whether any febrile alternations took place, Master L.'s feet were frequently felt, and they were found at times cold, and at other times of a dry heat. I have many times seen this disease, but the patients were too young, or too far advanced, to inform me, whether they had chillness succeeded by heat at its onset. 4. The disorders to which the young are more peculiarly liable afford a presumption, that hydrocephalus internus is an inflammatory disease; and this is confirmed by the regularity of the period, within which it finishes its course. And lastly, does it not happen more frequently than is suspected from external injury?

I have just now been well informed, that Dr. Rush has lately cured five out of six patients by copious bleedings. I relate here the reasons for an opinion without pretending to a discovery. Something like this doctrine may be found in certain modern publications, but it is delivered in that vague and diffuse style, which I trust your example will banish from medical literature."

Clifton, near Bristol,

July 28, 1795.

To this idea of Dr. Beddoes may be added, that the hydrocele generally succeeds an injury, and consequent inflammation of the bag, which contains it. And that other dropsies, which principally attend inebriates, are consequent to too great action of the mucous membranes by the stimulus of beer, wine, and spirits. And lastly, that as these cases of hydrocephalus end so fatally, a new mode of treating them is much to be desired, and deserves to be seriously attended to.

ADDITION [III]. On Vertigo.

To be placed after the additional Note at the end of Vol. I. on this Subject.

Having reperused the ingenious Essay of Dr. Wells on Single Vision, and his additional observations in the Gentleman's Magazine on the apparent retrogression of objects in vertigo, I am induced to believe, that this apparent retrogression of objects is not always owing to the same cause.

When a person revolves with his eyes closed, till he becomes vertiginous, and then stands still without opening them, he seems for a while to go forward in the same direction. This hallucination of his ideas cannot be owing to ocular spectra, because, as Dr. Wells observes, no such can have been formed; but it must arise from a similar continuance or repetition of ideas belonging to the sense of touch, instead of to the sense of vision; and should therefore be called a tangible, not a visual, vertigo. In common language this belief of continuing to revolve for some time, after he stands still, when a person has turned round for a minute in the dark, would be called a deception of imagination.