Diseases of the Horse's Foot

by H. Caulton Reeks

In the case of a deep but recent crack there is always more or less hæmorrhage. This favours risk of infection of the lesion with pus-forming organisms, and so leads to a more or less pronounced lameness, a degree of swelling, heat and tenderness in the coronet above, and a certain amount of surgical fever.

The acute symptoms subdued, but the fissure still remaining, gives us the crack we have classified as 'old.' This may in every case be distinguished from a more recent lesion by the amount of thickening of the overhanging coronet, and the presence of an increased quantity of sub-coronary horn in the region immediately about the crack. The previous inflammatory changes in the adjoining sensitive structures have here led to an increased secretion of horn, and a greater or less deposition of inflammatory connective tissue in the wounded coronary cushion.

Sand-crack of the toe always follows the direction of the horn fibres. That of the quarter, however, may on occasion run a course that is somewhat zigzag, first following the direction of the horn fibres for a short distance, then travelling in a horizontal direction, and finally continuing its course again in a line with the horn fibres, commonly at a point posterior to that at which it commenced.

In a quarter-crack that is old, and when contraction of the heels exists (which in this case it usually does), then will often be found overlapping of the edges of the crack. The expansion of the wall brought about when the body-weight is on the heels, cannot, by reason of the break in it, continue itself anterior to the crack. As a consequence, repeated expansion of the wall posterior to the crack, with the portions anterior to it in a state of enforced quiescence, leads in time to the posterior edge of the crack coming to lie over that of the anterior.

Complications.—The first complication likely to arise in a case of sand-crack is that attending simple laceration of the sensitive structures in a deep lesion. With the laceration all the phenomena of a repairing inflammation make their appearance. As a result, there is more or less heat according to the degree of inflammatory hyperæmia, swelling according to the amount of inflammatory exudate, and pain according to the amount of pressure the two foregoing bring to bear on the nerves in the inflamed area.

A second and more serious complication is the greater inflammation set up by the introduction into the crack of foreign substances. Small portions of gravel and flint, both by the irritation set up by their friction and by the infection they carry in with the dirt surrounding them, are responsible for the mischief.

When, from direct communication with the blood-stream, due to extensive hæmorrhage, bacteria from the outside gain entrance, this simple inflammation is further complicated by the formation of pus, or a limited gangrene of the keratogenous membrane.

In cases of great severity the gangrene of the keratogenous membrane spreads until the deeper structures are involved. We then get a necrosis (in the case of toe-crack) of the extensor pedis, and sometimes caries of the os pedis.

In like manner the necrotic changes occurring under these circumstances may invade the deeper structures in the region of quarter-crack. As a result of this, we may have the starting-point of suppurating corn, or necrosis of the lateral cartilage—in other words, cartilaginous quittor.

Commonly accompanying quarter-crack is the condition of contracted heels and atrophied frog. Sometimes described as a complication of sand-crack, it appears to us more rational to rather regard the sand-crack as a result or complication of the vice of contraction.