But there is another reason my daughter's teeth were not perfect, probably could not have been perfect no matter what we fed her, and why she will probably have at least some health problems as she ages no matter how perfectly she may choose to eat from here on. My daughters had what Dr. G.T. Wrench called "a poor start." Not as poor as it could have been by any means, but certainly less than ideal.
You see, the father has very little to do with the health of the child, unless he happens to carry some particularly undesirable gene. It is the mother who has the job of constructing the fetus out of prepartum nourishment and her own body's nutritional reserves. The female body knows from trillenia of instinctual experience that adequate nutrition from the current food supply during pregnancy can not always be assured, so the female body stores up very large quantities of minerals and vitamins and enzymes against that very possibility. When forming a fetus these reserves are drawn down and depleted. It is virtually impossible during the pregnancy itself for a mother to extract sufficient nutrition from current food to build a totally healthy fetus, no matter how nourishing the food she is eating may be. Thus a mother-to-be needs to be spending her entire childhood and her adolescence (and have adequate time between babies), building and rebuilding her reserves.
A mother-to-be also started out at her own birth with a vitally important stock of nutritional reserves, reserves put there during her own fetal development. If that "start" was less than ideal, the mother-to-be (as fetus) got "pinched" and nutritionally shortchanged in certain, predictable ways. Even minor mineral fetal deficiencies degrade the bone structure: the fetus knows it needs nutritional reserves more than it needs to have a full-sized jaw bone or a wide pelvic girdle, and when deprived of maximum fetal nourishment, these non-vital bones become somewhat smaller. Permanently. If mineral deficiencies continue into infancy and childhood, these same bones continue to be shortchanged, and the child ends up with a very narrow face, a jaw bone far too small to hold all the teeth, and in women, a small oven that may have trouble baking babies. More importantly, those nutrient reserves earmarked especially for making babies are also deficient. So a deficient mother not only shows certain structural evidence of physiological degeneration, but she makes deficient babies. A deficient female baby at birth is unlikely to completely overcome her bad start before she herself has children.
So with females, the quality of a whole lifetime's nutrition, and the life-nutrition of her mother (and of her mother's mother as well) has a great deal to do with the outcome of a pregnancy. The sins of the mother can really be visited unto the third and fourth generation.
This reality was powerfully demonstrated in the 1920s by a medical doctor, Francis Pottenger. He was not gifted with a good bedside manner. Rather than struggling with an unsuccessful clinical practice, Dr. Pottenger decided to make his living running a medical testing laboratory in Pasadena, California. Dr. Pottenger earned his daily bread performing a rather simple task, assaying the potency of adrenal hormone extracts. At that time, adrenaline, a useful drug to temporarily rescue people close to death, was extracted from the adrenal glands of animals. However, the potency of these crude extracts varied greatly. Being a very powerful drug, it was essential to measure exactly how strong your extract was so its dosage could be controlled.
Quantitative organic chemistry was rather crude in those days. Instead of assaying in a test tube, Dr. Pottenger kept several big cages full of cats that he had adrenalectomized. Without their own adrenals, the cats could not live more than a short time By finding out how much extract was required to keep the cats from failing, he could measure the strength of the particular batch.
Dr. Pottenger's cats were economically valuable so he made every effort to keep them healthy, something that proved to be disappointingly difficult. He kept his cats clean, in airy, bright quarters, fed them to the very best of his ability on pasteurized whole milk, slaughterhouse meat and organs (cats in the wild eat organ meats first and there are valuable vitamins and other substances in organ meats that don't exist in muscle tissue). The meat was carefully cooked to eliminate any parasites, and the diet was supplemented with cod liver oil. However, try as he might, Pottenger's cats were sickly, lived short and had to be frequently replaced. Usually they bred poorly and died young of bacterial infections, there being no antibiotics in the 1920s. I imagine Dr. Pottenger was constantly visiting the animal shelter and perhaps even paid quarters out the back door to a steady stream of young boys who brought him cats in burlap sacks from who knows where, no questions asked.
Dr. Pottenger's assays must have been accurate, for his business grew and grew. Eventually he needed more cats than he had cages to house, so he built a big, roofed, on-the-ground pen outdoors. Because he was overworked, he was less careful about the feeding of these extra animals. They got the same pasteurized milk and cod-liver oil, but he did not bother to cook their slaughterhouse meat. Then, a small miracle happened. This poorly cared for cage of cats fed on uncooked meat became much healthier than the others, suffering far fewer bacterial infections or other health problems. Then another miracle happened. Dr. Pottenger began to meditate on the first miracle.
It occurred to him that cats in the wild did not cook their food; perhaps cats had a digestive system that couldn't process or assimilate much out of cooked food. Perhaps the problem he had been having was not because the cats were without adrenal glands but because they were without sustenance, suffering a sort of slow starvation in the midst of plenty. So Dr. Pottenger set up some cat feeding experiments.
There were four possible combinations of his regimen: raw meat and unpasteurized milk; raw meat and pasteurized milk; cooked meat and raw milk; cooked meat and pasteurized milk, this last one being what he had been feeding all along. So he divided his cats into four groups and fed each group differently. The first results of Pottenger's experiments were revealed quickly though the most valuable results took longer to see. The cats on raw meat and raw milk did best. The ones on raw meat and pasteurized milk did okay but not as well. The ones on cooked meat and raw milk did even less well and those on all cooked food continued to do as poorly as ever.