ILLUSTRATIONS
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- Fig. 1—[Hoof Testers] 53
- Fig. 2—[Muscles of Left Thoracic Limb, Lateral View] 56
- Fig. 3—[Muscles of Left Thoracic Limb, Medial View] 57
- Fig. 4—[Sagital Section of Digit and Distal Part of Metacarpus] 59
- Fig. 5—[Ordinary Type of Heavy Sling] 62
- Fig. 6—[A Sling Made in Two Parts] 63
- Fig. 7—[Paralysis of the Suprascapular Nerve of Left Shoulder] 76
- Fig. 8—[Radial Paralysis] 78
- Fig. 9—[Merillat's Method of Fixing Carpus in Radial Paralysis] 79
- Fig. 10—[Contraction of Carpal Flexors, "Knee Sprung"] 95
- Fig. 11—[Pericarpal Inflammation and Enlargement Due to Injury] 99
- Fig. 12—[Hygromatous Condition of the Right Carpus] 100
- Fig. 13—[Carpal Exostosis in Aged Horse] 101
- Fig. 14—[Exostosis of Carpus Resultant from Carpitis] 102
- Fig. 15—[Distal End of Radius, Illustrating Effects of Carpitis] 102
- Fig. 16—[Posterior View of Radius, Illustrating Effects of Splint] 108
- Fig. 17—[Phalangeal Exosteses] 120
- Fig. 18—[Rarefying Osteitis in Chronic Ringbone] 121
- Fig. 19—[Phalangeal Exostoses in Chronic Ringbone] 122
- Fig. 20—[Contraction of Superficial Digital Flexor Tendon Due to Tendinitis] 138
- Fig. 21—[Contraction of Deep Flexor Tendon Due to Tendinitis] 139
- Fig. 22—[Chronic Case of Contraction of Both Flexor Tendons of the Phalanges] 140
- Fig. 23—[Contraction of Superficial and Deep Flexor Tendons] 141
- Fig. 24—[Contraction of Superficial Digital Flexor and Slight Contraction of Deep Flexor Tendon] 142
- Fig. 25—["Fish Knees"] 145
- Fig. 26—[Extreme Dorsal Flexion] 146
- Fig. 27—[A Good Style of Shoe for Bracing the Fetlock] 148
- Fig. 28—[The Roberts Brace in Operation] 149
- Fig. 29—[Distension of Theca of Extensor of the Digit] 151
- Fig. 30—[Rarefying Osteitis Wherein Articular Cartilage Was Destroyed] 153
- Fig. 31—[Ringbone and Sidebone] 156
- Fig. 32—[Position Assumed by Horse Having Unilateral Navicular Disease] 159
- Fig. 33—[The Hoof in Chronic Laminitis] 165
- Fig. 34—[Effects of Laminitis] 166
- Fig. 35—[Cochran Shoe, Inferior Surface] 168
- Fig. 36—[Cochran Shoe, Superior Surface] 169
- Fig. 37—[Hyperplasia of Eight Forefoot Due to Chronic Quittor] 176
- Fig. 38—[Chronic Quittor, Left Hind Foot] 177
- Fig. 39—[Skiagraph of Foot] 179
- Fig. 40—[Sagital Section of Eight Hock] 186
- Fig. 41—[Muscles of Right Leg; Front View] 187
- Fig. 42—[Muscles of Lower Part of Thigh, Leg and Foot] 189
- Fig. 43—[Right Stifle Joint; Lateral View] 190
- Fig. 44—[Left Stifle Joint; Medial View] 191
- Fig. 45—[Left Stifle Joint; Front View] 193
- Fig. 46—[Oblique Fracture of the Femur] 200
- Fig. 47—[Fracture of Femur After Six Months' Treatment] 201
- Fig. 48—[Aorta and Its Branches Showing Location of Thrombi] 210
- Fig. 49—[Thrombosis of the Aorta, Iliacs and Branches] 211
- Fig. 50—[Chronic Gonitis] 218
- Fig. 51—[Position Assumed in Gonitis] 219
- Fig. 52—[Spring-halt] 226
- Fig. 53—[Lateral View of Tarsus Showing Effects of Tarsitis] 228
- Fig. 54—[Right Hock Joint] 231
- Fig. 55—[Spavin] 235
- Fig. 56—[Bog Spavin] 243
- Fig. 57—[Thoroughpin] 247
- Fig. 58—[Fibrosity of Tarsus in Chronic Thoroughpin] 248
- Fig. 59—[Another View of Case Shown in Fig. 58] 249
- Fig. 60—["Capped Hock"] 252
- Fig. 61—[Chronic Lymphangitis] 258
- Fig. 62—[Elephantiasis] 259
INTRODUCTION
Lameness is a symptom of an ailment or affection and is not to be considered in itself as an anomalous condition. It is the manifestation of a structural or functional disorder of some part of the locomotory apparatus, characterized by a limping or halting gait. Therefore, any affection causing a sensation and sign of pain which is increased by the bearing of weight upon the affected member, or by the moving of such a distressed part, results in an irregularity in locomotion, which is known as lameness or claudication. A halting gait may also be produced by the abnormal development of a member, or by the shortening of the leg occasioned by the loss of a shoe.
For descriptive purposes lameness may be classified as true and false. True lameness is such as is occasioned by structural or functional defects of some part of the apparatus of locomotion, such as would be caused by spavin, ring-bone, or tendinitis. False lameness is an impediment in the gait not caused by structural or functional disturbances, but is brought on by conditions such as may result from the too rapid driving of an unbridle-wise colt over an irregular road surface, or by urging a horse to trot at a pace exceeding the normal gait of the animal's capacity, causing it to "crow-hop" or to lose balance in the stride. The latter manifestation might, to the inexperienced eye, simulate true lameness of the hind legs, but in reality, is merely the result of the animal having been forced to assume an abnormal pace and a lack of balance in locomotion is the consequence.
The degree of lameness, though variable in different instances, is in most cases proportionate to the causative factor, and this fact serves as a helpful indicator in the matter of establishing a diagnosis and giving the prognosis, especially in cases of somewhat unusual character. An animal may be slightly lame and the exhibition of lameness be such as to render the cause bafflingly obscure. Cases of this nature are sometimes quite difficult to classify and in occasional instances a positive diagnosis is impossible. Subjects of this kind may not be sufficiently inconvenienced to warrant their being taken out of service, yet a lame horse, no matter how slightly affected, should not be continued in service unless it can be positively established that the degree of discomfort occasioned by the claudication is small and the work to be done by the animal, of the sort that will not aggravate the condition.