Common Diseases of Farm Animals

by Robert Alexander Craig

A source of infection is by unknowingly buying cows that have reacted to the tuberculin test. The indiscriminate use and sale of tuberculin are largely responsible for the large number of reacting animals that have been placed on the open market. This dishonest practice has resulted in the rapid spread of the disease in certain localities. For years a large percentage of the breeding herds have been infected, and the writer has met with several herds of dairy and beef cattle that became tubercular through the purchase of tubercular breeding animals.

SYMPTOMS.—There is no one symptom by which we may recognize tuberculosis in cattle and hogs. None of the symptoms shown by a tubercular animal are characteristic, unless it is in the late stage of the disease. In a well-cared-for animal, the lymphatic glands in the different regions of the body, the lungs, liver and other organs, may be full of tubercles without causing noticeable symptoms of disease (Fig. 89).

[Illustration: FIG. 89.—A tubercular cow. This cow was, to all appearances, in good health, but showed generalized tuberculosis on post-mortem examination.]

Tuberculosis may attack any organ of the body, and in the different cases of the disease the symptoms may vary. Enlargement of the glands in the region of the throat, and noisy, difficult breathing are sometimes present. The udder frequently shows hard lumps scattered through the gland. Bloating may occur if a diseased gland in the chest cavity presses on the oesophagus and prevents the usual passage of gas from the paunch. Chronic diarrhoea may occur. If the disease involves the digestive tract, the animal is unthrifty and loses flesh rapidly. Coughing is not a characteristic symptom, and we should not place too much emphasis on it. If the lungs become tubercular the animal usually has a slight, harsh cough. The cough is first noticed when the cattle get up after lying down, when the stable is first opened in the morning and when the animals are driven. If the chest walls are thin, soreness from pressure on the ribs may be noted. By applying the ear to the chest wall and listening to the lung sounds, absence of respiratory murmurs and abnormal sounds may be distinguished, due to consolidation of the lung tissue, abscess cavities and pleural adhesions. In a well-advanced case the hair is rough, the skin becomes tight and the neck thin and lean. The animal may breathe through the mouth when it is exercised. Weakness may be a prominent symptom.

[Illustration: FIG. 90.—Tubercular spleens.]

Breeding animals that are well fed and cared for may live for several years before showing noticeable symptoms of tuberculosis. The disease progresses more rapidly in milch cows, especially if given poor care. Calves allowed to nurse a tubercular mother that is giving off tubercle bacilli frequently develop enlarged throat glands and the intestinal form of the disease.

Hogs develop a generalized form of tuberculosis more quickly than cattle, but an unthrifty, emaciated condition is seldom noted in hogs under ten months old.

POST-MORTEM LESIONS.—The effect of the tubercle bacillus on the body is to irritate and destroy the tissues. Lumps or tubercles form in the lymphatic glands, liver, lungs, spleen (Fig. 90), serous membranes, kidneys and other body organs (Figs. 91 and 92). The tubercles may be very small at first, but as the disease progresses they continue to enlarge until finally a tubercular mass the size of a base-ball, or larger, is formed (Figs. 93, 94, 95 and 96). Lymphatic glands may become several times larger than normal and the liver and lungs greatly enlarged. The pleura and peritoneum may be thickened and covered with tubercles about the size of a millet seed, or larger. Pleural and peritoneal adhesions to the organs within the body cavities are common.

[Illustration: FIG. 91.—The carcass of a tubercular cow. Note the condition of the carcass, and the tubercular nodules on the chest wall, showing that the disease was well advanced.]

[Illustration: FIG. 92.—A section of the chest wall of a tubercular cow showing a better view of the diseased tissue.]