In labor the anterior wall of the vagina is so depressed, stretched, and shortened by the advancing head that during and after the second stage the anterior lip of the cervix may be seen behind the urethra. If the puerperium progress favorably, with prompt involution of the uterus, vagina, perineum, and peritoneum, the relaxation of the vesico-vaginal wall and of the utero-sacral supports disappears and the uterus resumes its normal multiparous location and position.7 But if the enlarged uterus remain in the long axis of the vagina, with its fundus incarcerated in the hollow of the sacrum between the utero-sacral ligaments, and with its sacral supports so stretched that they cannot recover their contractile power, and with involution of all the pelvic organs arrested, the descent may not only persist, but may even progress with constantly increasing cystocele to the third degree of prolapse. The downward influence of the above conditions may be materially increased by rupture of the perineum, and consequent prolapse of the recto-vaginal wall into a pouch called rectocele.
7 The anteflexion of the multiparous uterus is less than that of the virgin.
In the great majority of cases of complete prolapse the posterior vaginal wall in its descent is peeled off from the rectum, leaving the latter in its normal position. In rare instances the lower portion of the rectum is also found to have extruded in extreme rectocele, making a pouch below and in front of the anus, where fecal matter may accumulate and remain in hard scybalæ.
Obviously, complete prolapse of the uterus is only an incident to the prolapse of the pelvic floor. The whole mechanism is in all respects analogous to that of hernia. The extruded mass drags after it a peritoneal sac, which, hernia-like, contains small intestine. This sac forces its way to the pelvic outlet and extrudes through the vulva, having the inverted vagina for its covering.
| FIG. 3. |
| First Degree of Prolapse of the Post-partum Uterus. The posterior vaginal wall has been changed from its normal forward direction to a vertical direction by perineal rupture and anterior displacement of the cervix; the vesico-vaginal wall descends in cystocele, becomes hypertrophied, and drags the heavy uterus after it. The descending uterus carries with it a reduplication of the vaginal walls. |
In descent of the first degree the location of the uterus is either changed to a lower level, the position remaining normal, or, as is more common, the cervix having moved nearer to the symphysis and the organ turns back into retroversion. In a given case suppose the vaginal walls from some cause to have become relaxed and to have settled to a lower level in the pelvis. As an associated fact the uterus to which these walls are attached must then also occupy a place correspondingly nearer to the vulva—i.e. the location of the uterus has changed, so that space enough intervenes between it and the hollow of the sacrum for the former to turn back into the position of retroversion or retroflexion. If, on the contrary, the descending uterus still maintains its normal anteversion and anteflexion, it must occupy space which belongs to the bladder. The vesical irritation consequent upon this mal-location has generally been ascribed to the anteversion and anteflexion, which are therefore oftentimes wrongly pronounced pathological. The prompt relief which follows permanent replacement of the organ in the normal location, even though in so doing its anteposition be exaggerated, proves that the symptoms depend upon the mal-location, not upon the anteposition. The importance of a clear distinction, therefore, between location and position becomes apparent. Vesical irritation, moreover, is sometimes caused by the dragging of the uterus upon the neck of the bladder. This traction occurs not only in ascent, but also when the organ descends below a certain level.
| FIG. 4. |
| Showing Extreme Descent of the Uterus and of the Pelvic Floor, and the Hernial Character of the Lesion. |
In the foregoing paragraphs traction due to the falling pelvic floor has been discussed as a cause of descent. The impairment of the uterine supports may, however, be such that instead of falling and dragging the uterus after them, they simply permit it to descend along the vaginal canal by the force of its own weight, and to carry with it the reduplicated vaginal walls. This influence is generally enforced by the increased weight of the diseased organ. The vagina more readily becomes a track for the descending uterus when from any cause the normal forward direction of the vaginal canal changes toward the vertical: this change may occur either as the result of a forward displacement of its upper extremity, involving anteposition of the cervix, or of a retro-displacement of its lower extremity in consequence of rupture or subinvolution of the perineum. (See Fig. 3.) Descent in the track of the vagina is obviously combined with some degree of retroversion, because the axes of the uterus and vagina then correspond.