A sailor, 41, got hygroma of the right knee in 1915, was operated on in July, returned to his dépôt a month later, and thence to Vizille Urage by reason of contracture in extension of the right leg. It was thought he was simulating (since there was no muscular atrophy), and he was sent to the neurological center, where under anesthesia the joint was found free. This man developed, when the knee was bent, extraordinary cracklings in the joint, and he showed pain unequivocally, making a defensive movement, partly reflex, partly voluntary, when the leg was flexed beyond a certain point. There was 3.5 cm. atrophy in the thigh, a reflex atrophy due to the joint disorder. There were no other signs of hysterotraumatic contracture.
According to Sollier, the diagnosis of hysterotraumatic contractures depends upon: first, a characteristic special attitude of the contractured limb; secondly, the participation of the antagonists as a group (global); thirdly, the superposition of sensory disorder upon motor disorder (Charcot’s law); fourthly, the segmentary topography of sensory disorder; fifthly, the extension of the contractured joint; sixthly, the persistence of the contracture in the same form, whether at rest or in attempted movements; seventhly, muscular rigidity; eighthly, normal tendon reflexes; ninthly, normal electrical reactions (though R. D. is hard to determine in muscles contracted to the maximum); tenthly, special reactions during attempts to reduce, such as pains, and equal and regular resistance to changed attitude, pseudoclonus in cases of foot contracture; eleventhly, immediate reproduction of the contracture after reduction under chloroform; twelfthly, co-existence of various hysterical stigmata.
Crural monoplegia, tetanic. Recovery.
Case 391. (Routier, 1915.)
An ensign was wounded by a shell splinter in the right scapular region September 25, 1915. A large hematoma was drawn off and drains inserted. Antitetanic serum was given 24 hours after the trauma. The wound looked well. The patient complained merely of the heaviness of his arm, and after September 27, the temperature fell to normal. Magnesium chloride solution was applied every other day, and progress was so good that evacuation was ordered.
However, October 8, the patient suddenly began to complain of a sharp pain in the right thigh, which next day became intolerable and threw the muscles into a slight contracture, the adductors being extremely stiff. Headache developed in the course of the day, with slight stiffness of neck, exaggeration of reflexes in the right leg, and ankle clonus. Temperature: 37.6 morning, 38.5 evening. The patient was isolated and given chloral.
October 10, paroxysmal crises of pain, more marked stiff neck, and lumbar stiffness appeared, with nervousness, photophobia, and hyperesthesia to noise. The wound seemed to be doing well. Chloral was given.
Slight trismus developed October 11. The tongue became dry and the patient drank little. The condition held and the same treatments were repeated up to October 15, when the temperature fell and the contractures and pains were diminished. The chloral was continued. There were still a few cramps in the neck. October 22, however, the patient was practically well.
We are here dealing with an instance of local tetanus of monoplegic form, developing a fortnight after the wound (there is an early group developing, as a rule, from the fifth to the tenth day, and a group of later development, after the twentieth day; the interval in this case was of intermediate duration). According to Courtois-Suffit and Giroux, the differential diagnosis is not easy, since, besides tetanus, must be considered tetany, spastic monoplegia of cerebral or spinal origin, partial hemiplegia, peripheral neuritis, contractures due to bone, joint, muscle or tendon lesions, strychnine intoxication and hysterical contractures. Three cases out of six described by Routier were fatal.